Coconut oil contains many healthy fats; it is obtained from coconuts, which contain medium chain fatty acids. Coconut oil is a popular cosmetic product. Coconut oil is obtained from the fruit of the coconut tree, the coconut. Coconut oil is widely used for cosmetic purposes (it is applied to the skin as a natural moisturizer). The effects of this oil on the skin and hair after ingestion are not yet known. The majority (65%) of coconut oil consists of medium chain triglycerides (MCTs), a class of substances that includes triglycerides and fatty acids with a carbon chain of 6 to 12 chains. Research has shown that if you replace ordinary fats with MCTs (as your main source of calories) while staying within the calorie tolerance of your daily diet, your body will begin to burn fat faster over time. In general, this effect is slightly more pronounced in obese people. In addition, taking coconut oil speeds up the metabolism (for a while) and begins to break down fat faster, resulting in the release of useful fatty acids (lipolysis). This effect is observed at the beginning of the intake of coconut oil and usually disappears after 2 weeks. During the decomposition of coconut oil, the body produces more ketone bodies than long-chain fatty acids. According to one study, this explains the ability of coconut oil to maintain muscle mass in obese people on a strict hypocaloric diet. This does not apply to slim people. It is unlikely that including coconut oil in your diet will result in significant weight loss, but the beneficial fatty acids in this oil will undoubtedly provide a better alternative to other dietary fatty acids, enriching your diet with valuable micronutrients. Other names: coconut, coconut oil, medium chain triglycerides (partial synonyms, but in practice refer to the same concept).
Class of substances: Ayurveda products Food combined with : Vitamin E (which is better absorbed when SCT is used as a base beforehand). You can get the maximum benefit from coconut oil by including 5 to 10 g of medium chain triglycerides (about 7.7 to 15 g of coconut oil) in your daily diet. Coconut oil can be used for cooking but should never be heated to a smoking temperature (350degF/175degC). Substitution of other edible fatty acids with coconut oil may reduce their fat-dissolving effects if the caloric value of the oil is greater than the energy value of these fatty acids. Sources and Composition Sources The term “coconut oil” is used to describe a vegetable oil obtained from the coconut (the fruit of the coconut palm which belongs to the palm family1) and is frequently used in cooking because of its high content of valuable fatty acids and consequent health benefits, and of course the unique flavor of the coconut; Coconut oil is also used in soaps, cooking fats, chocolate, lollipops, candles and bedside lamps. Coconut oil is obtained from copra (dried coconut pulp), which consists of 60-70% fatty acids, 4-10% water, protein (less than 10%) and non-sugar carbohydrates (less than 20%); the fatty acids are bleached and deodorized (odorless) to produce odorless and tasteless extra virgin coconut oil. If this step is skipped and the fatty acids of the coconut pulp are used as raw material, the result is second-pressed coconut oil (which is more commonly used in cooking).
As for the cosmetic use of coconut oil, it is hydrogenated, which modifies its physical and chemical properties (notably by increasing its melting point). Coconut oil has been used for centuries in traditional Indian medicine, especially in Ayurveda where the term Kalpa Vriksha is used to designate the coconut tree. The oil is commonly used to treat alopecia, burns and diseases of the cardiovascular system, intestinal worms, gonorrhea, bronchitis, gingivitis and as an antipyretic. Coconut oil is formed by the fatty acids of the coconut, so it has long been used for cosmetic purposes. In addition, it is an excellent natural antiseptic and has a wonderful taste.
Composition Coconut oil contains the following fatty acids: Note: The names of the fatty acids refer directly to the number of carbons in the side chain followed by a number of unsaturated carbons; for example, “14:2” means 14 carbons in the side chain and 2 unsaturated carbons. All fatty acids named “x:0” (without unsaturated carbons) are saturated fatty acids, and those with 6 to 12 carbons in the chain are medium-chain triglycerides (MCT). caprylic acid, 8%; capric acid, 7%; lauric acid (49%2) – Saturated fatty acid (SFA) has 12 carbon atoms and the chemical name is “12:0”; myristic acid (17.5-18%) – SFA has 14 carbon atoms (14:0); palmitic acid (8-9%) – SFA has 16 carbon atoms (16:0); stearic acid (2-3%) – SFA has 18 carbon atoms (18 : 0); butyric acid (5-6%) as monounsaturated fatty acid omega-9 (18:1); linoleic acid (1.8-2%) and omega-6 (18:2); In addition to fatty acids, coconut oil contains: vitamin E (tocopherol); vitamin C – 15mg/100ml of coconut water; sterols; squalene Of the fatty acids listed above, 90% are saturated fats, 7% are monounsaturated fatty acids (mainly butyric acid), and the remaining 3% are linoleic acid and traces of alpha-linoleic acid. Of these fatty acids, approximately 65% are medium chain triglycerides. Triglycerides (three fatty acids attached to a glycerol “frame”; the standard form of fatty acids) are generally represented in coconut oil by trimyristin, trilaurin, tripalmitin and tristearin. The triglyceride composition of coconut oil can change when it is mixed with other oils (a process called “over-esterification”).3) Up to 90% of the fatty acids in coconut oil are mainly medium-chain saturated fatty acids (which represent about 65% of the total fatty acids in coconut oil).
Neurology
Appetite
In an experiment (to examine the relationship between fatty acid length and hunger) in healthy, lean men who were given dairy products (short-chain fatty acids), coconut oil (medium-chain fatty acids) or lard (long-chain fatty acids) of equal caloric value, the researchers observed no difference in the appetite of these men. At present, due to the lack of scientific evidence, it is not possible to prove the appetite suppressant properties of coconut oil (fatty acids have a natural satiating effect, so it is not certain that these properties are more pronounced in coconut oil than in other oils). Pain The results of pain tests with a hot plate indicate that both acetic acid (formol) and coconut oil (in proportion to the amount) have analgesic effects. 2.3 Amyotrophic Lateral Sclerosis (ALS) and Information Processing Speed An experiment involving healthy adults who consumed 1200 mg per day of coconut oil (as a placebo fish oil) for 4 weeks improved eye-hand coordination test results, but not those who took fish oil4. )
According to ALS-Untangled (a peer-reviewed panel of experts), coconut oil would disrupt mitochondrial complex I, as demonstrated in laboratory studies (cetonic bodies are incubated outside the living body with CMT, which in humans occurs in neurodegenerative diseases such as ALS; 5) according to the authors of the study, lipid metabolism is activated when calories are ingested (against ALS). “The authors conclude that a direct correlation between coconut oil and the aggravation of ALS has not been established (in 2012), and that only in rodents (for which data are scarce) does a high-fat diet (cetogenic diet) inhibit the development of ALS. As we can see, due to a lack of scientific evidence, scientists are still not able to prove that coconut oil prevents the development of ALS. Cardiovascular health Triglycerides and lipoproteins According to a recent meta-analysis (which compared the results of prospective epidemiological studies), triglycerides and lipoproteins are essential elements of cardiovascular health.
studies that investigate the role of fatty acids in their own and in the everyday life of human beings) and the consumption of fats does not alter the cholesterol levels in blood. This could be due to the wide range of fatty acids that are found in coconut oil. These include it contains lauric acid (which is responsible for more than 50% of the calories within coconut oil) together with myristic acid, as determined by researchers, increase HDL and lower LDL in blood (the conclusion came from brief review and health reports) Coconut oil improves HDL-C (according to research) in blood, but without reducing the levels of LDL-C. However, coconut oil can increase the level of LDL-C present in the blood, which may result from its higher content of myristic and caprylic acids.6 ) Reviewing the results of studies that examined the cholesterol levels with low density (LDL-C) levels in blood samples of people who consumed 30 milliliters (270kcal) or 270kcal of coconut oil in the daily (hypocaloric) eating habits (whereas the people who were in control consumed soy oil) Researchers observed an 8.2 percentage increase in blood HDL levels in the prior group, which was not seen in the group not monitored, and there was a normalization in ratios of LDL as well as HDL in the second group, while those who ate the soybean oil (the overweight women) were more susceptible to have higher overall and LDL cholesterol levels. If coconut oil was included into the high fat food regimen (38 percent calories however with different fat acids) Participants in the first group showed higher levels of HDL-C (by 17.3 percent) and also had lower levels of cholesterol levels of LDL (by 2.5 per cent) and had lower levels of the LDL cholesterol (by fourteen percent) However, this wasn’t the case with the group in the control who consumed the mixture of polyunsaturated and monounsaturated acids. People who ate the foods that were high in lauric acid also had lower blood cholesterol levels overall (relative to trans fat) but not as much as those who consumed palmitic acids, butter (which is effective in lowering HDL-C levels as well the levels of LDL-C) and beef tallow, and the sunflower oil.7 ) Contrary to the study previously conducted, another investigation (20 percent of the participants consumption of calories per day was comprised of oils) discovered that soybean oil reduced blood cholesterol more effectively than coconut oil (which increased the level of the LDL-C) However the study did not show any effect on HDL-C levels for either of the groups. Plantains were added to the diet, blood cholesterol levels decreased in the same way for everyone, regardless of the type of oil they consumed. It is thought that coconut oil helps to control the levels of LDL and HDL in the body and thus can have an impact on cardiovascular health , as evidenced by the limited number of studies on this subject. In the event that coconut oil gets used to the food plan of a day , it is observed that amounts in HDL and LDL-C are elevated, but there is evidence suggesting that this happens more with HDL-C. This is typical of research regarding diets and food items. They’re conducted with no placebo controls (another fat acid can be an alternate to placebo) so the results of these studies aren’t considered as valid. In a study of patients with diabetes who consumed 18g of medium chain triglycerides (vs. 18g of long-chain triglycerides) each day for the course of 90 days the subjects’ cholesterol level fell (by 12 percent) in addition to HDL-C (by 17 percent)) along with the LDL-C (by 17.5%) and HDL-C (by 16 percent) However the levels of triglycerides did not change. unaffected.8 ) Responses of the glucose metabolism. A 90-day study involving type II diabetics given 18g of medium-chain triglycerides each daily (the results of which were contrary to the effects of long-chain triglycerides weighing 18g) that resulted in a slight decrease in the body weight of the participants and an improvement in levels of insulin resistance (by 17 per cent) according to the homeostatic test for insulin resistance. The group in control suffered from deterioration (increased to 7.3 per cent) and the distinction is massive despite the fact that the variables weren’t identical initially, and sugar levels at the time of fasting did not change throughout the test. Obesity-related processes and the fat-mass ratio Medium-chain Triglycerides (which comprise 65 percent of coconut oil as a percentage in weight) are more prone to oxidation than longer-chain acids, as evidenced in animal studies that examined the use of substrates) and in research that involve humans.10 ) For humans , consumption can trigger active oxidation of long-chain fats (not forgetting medium-chain fats) and, as per researchers it is important to consider this as a weight-loss strategy since overweight individuals are more likely to undergo oxidation of long-chain fatty acids in a slower manner than their slim counterparts even the fact that FFAs are processed in the normal rate in each. This seems to be due to the activity of the enzyme carnitine palmitoyl-transferase (PPT), which prevents and slows down the oxidation of fatty acids and does not “need” medium- and short-chain fatty acids at all.
Medium chain triglycerides (65 percent of coconut oil) are more quickly processed when it comes to fat-burning (fat breaking or burning fat) instead of longer-chain acid fatty acids. It’s something to consider for those who are overweight and have less oxidation in long-chain acids. Studies of over-fed (deliberately) animals have shown that the consumption of medium-chain fatty acids won’t produce the same weight gain as long-chain acids, despite the fact that animals are overweight.11 ) Coconut oil (40 percent of calories ) fats (80 percent of which of which is made up from coconut oil) as well as long chain fats (same amount found in meat cholesterol) was consumed over 14 days by the group that was not consuming. The metabolic rate rising by 4.3 percent in those who took coconut oil (by day 7) However, this effect was gone after end of the study and similar results were observed in a different study that involved females in good health. It is evident that coconut oil can boost metabolism, but it is only for a short period of time (the effect doesn’t become apparent until two weeks after the consumption by the oil) this means that it’s not possible to talk about an ongoing weight loss (the effects of which last for a long time). Ketogenesis is a prevalent issue for people who are overweight (BMI over 30) who consume medium chain triglycerides (9.9g CCT) in an eating pattern that is hypocaloric (578.4 kcal/day) increases the ketone body (beta hydroxybutyrate) in blood. This also hampers the elimination in nitrogen out of the human body. This , according to studies, a trigger for the growth of proteins in cells. The participants in the study lost weight in only two weeks. Most of the fat that was in the body (56 percent) resulted from eating foods that have longer chain (22 per cent) or foods that were very low in calories (25 percentage). This chapter is focused on the features that coconut oil (which is of 65%) medium chain fat often referred to MCA) and diet-based MCAs. If long-chain fatty acids are used to regulate activities in the study, the term DCH is used to refer to the fat acids. In obese people, supplementation with DCAs resulted in active oxidation of fat acids, which is similar to overweight people (BMI=25-33) and isn’t most likely to cause the reason of the weight loss after six months (40 per cent of diet and 75% were oil-based fats) as well as olive oil was utilized as a means of controlling in the study case.12) Experimental study of women with central Fatty Acids obesity , who consume thirty milliliters (270cal) from coconut oil, or 30ml controlled oil (soybean oil that has a of 270 calories) every day for 12 weeks while adhering to the hypocaloric, high-carbohydrate-based diet and regular exercise, showed that even though they experienced similar weight loss and reduction in BMI and reduction in waist circumference, the waist circumference decreased (by 1.4cm) within the first group of participants taking coconut oil. Similar results were observed in another study where the participants (BMI=24.6-24.7) were given 1.7g of coconut oil each day for 12 weeks (while those who were in the group that had a control were given different oils) that resulted in each group losing weight, while shrinking their waists, however, the results were evident more so when they consumed CCT. A different study revealed that the participants consumed 10g SCT (group 1) and 10 grams of DTT (group two) DTT (group two) every day for 12 weeks in eating a diet (2,200+60 grams of fats per day) The study found that those in the group 1 (BMI>23) gained weight more (3.86+/-0.3 kg) toward the end of the study than those who ate DTT (BMI>23; 2.75+/-0.2kg) and those who had a lower BMI (<23) didn’t experience no significant weight loss.
This may be due to decreased oxidation of long-chain fat acids in obese people that is normalized through the use of CCT. A different study showed that participants ate a specific type of bread daily for twelve consecutive months (14g of fats and 1.7g of CCT) to CCT) resulted in greater reductions in body weight of participants (by 6.1+/-0.5 percentage) when compared to those in the group the research who consumed DTT throughout the study (by 4.5+/-0.5 percentage) Another study included type II diabetics taking 18g of CCT every day for 90 days resulted in normalized hemoglobin glucose (A1c) levels and also a decrease in weight for the subjects (by 2.6 percent between 132 pounds to 128.6 pounds on average) but no change was observed in the case of people with diabetes (who had been taking DTT). 13 ) As you can see, coconut oil, and specifically DTCs may be more efficient when it comes to breaking down fats inside the body opposed to other oils, which is particularly true for obese people, not just slim people. Coconut oil’s benefits aren’t “overwhelming” (and in some tests, participants did not shed weight anyhow). Bioenergetics of Endurance as well as Physical Activity According the researchers, generally, the process of replacing long-chain fats with medium chain triglycerides is that indicators such as physical and endurance don’t change [4949] (or in the case it does, the effect isn’t as favorable) regardless being that, according to the theories of things PTCs are faster catabolized. This results in the body turning into fatty acids as “fuel” during exercise.14 ) When studying the effects of glycogen, research has concluded glycogen does not affect endurance of those who exercise aerobically (running and walking) over large or moderate distances regardless of any other consumption of carbohydrates. As you will see, there is numerous solid and reliable evidence to show that the energy are derived from medium-chain triglycerides, as along with coconut oil are a “better” source of energy during exercise than long-chain fat acids regardless of the fact that calories be ergogenic (help in improving performance).
Study of the mechanisms of inflammation and immunity of rats who received coconut oil for the treatment of inflammation found that it has an anti-inflammatory effect, which reduces the symptoms of acute inflammation (inflammation type, carrageenan-inducing foot swelling) However, rats who suffer with chronic inflammation didn’t suffer any adverse effects from using coconut oil (in the granuloma study using an swab of cotton). It’s not too far to mention the study conducted with rats in which the IL-6 (basal and epinephrine-driven) was released from fat cells. The rats were fed two different oils: coconut or olive, which did not result in an increase in the level of epinephrine-induced I-6 in the blood of their subjects (although the levels of IL-6 among rats who received coconut oil was more susceptible to epinephrine-induced IL-6 than that of the second group) however, for the group who received sunflower oil and IL-6 levels that were present in blood, despite being at first low, increased over the course of the course of the study (in response to the effects on the epinephrine). (15) The anti-inflammatory property localized of coconut oil were apparent in an experiment involving people who, after receiving the oil, experienced the waist circumference shrink regardless of changes in body weight or weight. It is thought to be an indicator of chronic inflammation which is of low-grade. The study that involved participants being served muffins made from coconut oil in breakfast samples revealed that, in contrast to the fish oils and Linseed oil (which in various ways can affect the levels of IL-8 present in blood) coconut oil had no effect upon that parameter.16) The results are apparent that, as we can see contrary to other fats, coconut oil is anti-inflammatory impact (weakly visible). The reaction to organ systems cavities made of coconut roots has been utilized for dental hygiene and for washing since the time of the ancient times due to its low toxicity as well being anti-inflammatory properties (due because of the lauric acid). 17.) 17.) Coconut (the fibers that make up the nuts) has antibacterial properties and is able to combat the different bacteria and viruses present in the mouth. It is believed that this comes due to the presence of sucrose monolaurate sugar. Researchers have found that it reduces the effects of oxidation on Streptococcus mutagens (at 0.05 0.5 percent) and also decreases the amount of plaque that living creatures develop. It has also been demonstrated through a study that involved humans (although it’s uncertain about the less effective) that used coconut soap to soften their teeth, and protect the mouth from stomatitis. 18 18) Coconut shell fibers have antibacterial action, preventing oral bacterial infection and lesions; the oil itself has this same effect (as was proven in another experiment), but it is rarely used for oral lubrication (it is usually swallowed) unlike coconut shell fibers, which are chewed like “gum. Liver Against a background of liver damage from paracetamol, taking 10ml/kg of coconut oil stops liver enlargement (100mg/kg of the comparison drug, silymarin in sea thistle , also proved effective in this case), whereas a lower dosage (1-5ml/kg) of coconut oil fails; similar changes occurred with liver enzymes in serum and liver histology. This does not appear to be related to the antioxidant properties of coconut oil, as they were not confirmed in an 8-week experiment with rats whose diets included 15% coconut oil daily. Unlike other oils (copra, olive oil, and sunflower oil), coconut oil is effective inhibe la formation de graisses dans le foie, comme cela a ete demontre chez des rats ayant recu 8 % d’huile de coco par jour pendant 45 jours, ce qui a entraine une desactivation de l’hydroxy-methylglutaryl coenzyme-A dans les cellules du foie de ces rats, tandis que la lipoproteine lipase (LPL), en revanche, devient plus active. L’huile de coco reduit la LPL hepatique (mais n’a aucun effet sur l’HMG-Co-A) et active l’HMG-Co-A reductase tout en augmentant l’efflux d’acide biliaire, ce qui permet d’obtenir un “effet hypocholesterolemique” absolu, et chez les rats nourris aux graisses, 40ml/kg d’huile de coco reduit les niveaux de cholesterol hepatique aussi efficacement que 0,1mg/kg de lovastatine.19 ) Comme nous pouvons le constater, l’huile de coco (en grande quantite) protege le foie des dommages toxiques (selon des donnees preliminaires), tandis que chez les rats, l’ingestion prolongee d’huile de coco et de liquide de coco ralentit la synthese des lipides et favorise leur degradation, ce qui est benefique pour le foie en general. Reactions capillaires esthetiques En Inde, les femmes utilisent l’huile de noix de coco (ainsi que l’amla et l’huile de moutarde) depuis des siecles pour renforcer et faire pousser de beaux cheveux epais. L’huile de coco est connue pour penetrer dans les follicules pileux lorsqu’elle est appliquee sur les cheveux, ce qui renforce les cheveux de la racine a la pointe et les protege des dommages mecaniques (peignage, organismes vivants exterieurs), contrairement aux huiles minerales et de tournesol qui, lorsqu’elles sont appliquees sur les cheveux, “arrachent” les proteines ; L’effet protecteur de l’huile de coco est valable aussi bien pour les cheveux normaux que pour les cheveux decolores (mais pas pour les cheveux “brules”) et il est preferable d’appliquer l’huile juste avant un “facteur de stress” (brushing, etc.). d. Ces proprietes de l’huile de coco ont ete prouvees lors d’essais sur l’homme evaluant l’indice de sante des cheveux (WHI) en appliquant quotidiennement de l’huile de coco sur les cheveux pendant 16 semaines, ce qui a permis de reduire considerablement la fragilite des cheveux et d’eliminer les pointes fourchues et seches (une cause majeure de casse des cheveux). 20) En etudiant les follicules pileux ou les meches de cheveux individuels, les scientifiques ont decouvert que l’huile de noix de coco rend nos cheveux plus hydrates en emprisonnant cette humidite (contrairement a l’huile minerale) a travers un film “huileux” invisible sur chaque cheveu. L’huile de coco peut etre utilisee comme shampooing, elle n’irrite pas le cuir chevelu (et ne pique pas si elle entre en contact avec vos yeux). Comme nous pouvons le constater, l’huile de coco protege nos cheveux des dommages et les hydrate (et retient cette humidite, creant ainsi un film protecteur a la surface de chaque cheveu). En outre, l’huile de coco est souvent ajoutee aux shampooings (les proprietes physiques, la viscosite et la solidification de l’huile a temperature ambiante doivent etre prises en compte). Peau Lors de l’evaluation de l’incidence de l’irritation de la peau (lors de l’application topique de l’huile), les chercheurs ont interroge 480 personnes souffrant d’affections cutanees avancees, parmi lesquelles seules 5 personnes (0,9%) ont eu une reaction allergique sur la peau lors de l’application de 15ul d’une solution de 5% de cocoate de potassium avec un coton, 12 personnes etaient allergiques a la cocamidopropyl betaine, alors que la solution d’huile de coco a 100% n’a provoque aucune reaction allergique chez quiconque. 21) Aucune allergie a l’huile de coco n’a ete constatee chez les animaux ou les humains (meme ceux qui sont allergiques a la cocamidopropyl betaine) qui ont applique sur leur peau de l’acide laurique isole et l’huile elle-meme. L’huile de coco, lorsqu’elle est appliquee sur la peau, ne provoque pas d’allergies ou de reactivite immunitaire cutanee et est donc ideale pour les personnes ayant une peau sensible ou irritee. Chez les personnes atteintes de xerose (peau pathologiquement seche et irritee) qui doivent hydrater activement leur peau, l’huile de noix de coco (ainsi que l’huile minerale de controle) rend la peau plus hydratee. L’effet hydratant de l’huile de coco a ete confirme dans une experience (methode en aveugle) impliquant des adultes atteints de dermatite atopique, qui ont applique 5 ml d’huile de coco (deux fois par jour) sur les zones cutanees affectees, ce qui s’est avere plus efficace que l’huile d’olive (huile de controle) pour reduire l’indice SCORAD (de 46,8% par rapport aux valeurs initiales) ; Sur 20 personnes presentant des lesions cutanees infectieuses a Staphylococcus aureus (dont l’environnement benefique est la dermatite atopique) qui ont applique quotidiennement de l’huile de coco pendant 4 semaines sur les zones affectees, la bacterie n’a survecu que chez une seule. 22 ) L’huile de coco hydrate la peau et aide a combattre la dermatite atopique, grace a ses proprietes antibacteriennes et a ses proprietes hydratantes. Une experience menee avec des rats auxquels on a applique de l’huile de coco quotidiennement pendant 10 jours sur leurs plaies (premiere application apres 24 heures apres une lesion cutanee), a entraine une acceleration de la cicatrisation (ce qui n’est pas le cas avec l’huile temoin). Le monolaurate de glycerol (un composant gras de l’acide laurique adjacent au glycerol), parfois aussi appele lauricidine, est aussi efficace (pour combattre la bacterie nuisible serratia marcensis, lorsqu’il est applique sur les mains) qu’une solution isopropylique alcoolique a 70 % (avec seulement 1,5 % de lauricidine necessaire). Appliquee par voie topique sur les zones affectees par la dermatite atopique, l’huile de coco reduit le nombre de Staphylococcus aureus. L’huile de coco est souvent ajoutee aux savons liquides en raison de ses proprietes antibacteriennes (qui, selon les scientifiques, sont dues a sa teneur en monolaurate). Sexe et grossesse Avantages pour l’enfant a naitre L’huile de coco est censee assurer une croissance et un developpement corrects des nouveau-nes grace aux acides gras qu’elle contient. En outre, il est utile de frotter les bebes avec de l’huile de noix de coco, qui procure une agreable sensation tactile au contact de la peau delicate du bebe et constitue un hydratant naturel. 23) Si les nouveau-nes sont regulierement masses avec de l’huile de coco (4 fois par jour a partir du deuxieme jour de vie jusqu’a l’age d’un mois), ils prendront du poids plus rapidement et leur longueur corporelle augmentera (on ne peut pas en dire autant des massages avec de l’huile de controle). Ni l’huile de coco ni l’huile minerale n’affectent les reactions neurocomportementales des nouveau-nes. Comme nous pouvons le constater, un massage regulier a l’huile de coco accelere la prise de poids des nourrissons. Reactions biogenes de l’ALC L’acide linoleique conjugue (ALC) est un acide gras dont les scientifiques pensent qu’il reduit la graisse corporelle, mais cela n’a pas encore ete prouve. La perte de poids induite par l’ALA chez les souris est acceleree lorsque l’huile de noix de coco est administree en meme temps que l’ALC (ce qui n’est pas le cas avec l’huile de soja temoin), et les effets de l’huile de noix de coco sont etendus aux souris dont le regime alimentaire est exempt de graisses ; 24) certains rapports indiquent que l’association du CLK et de l’huile de coco est plus efficace pour reduire le surpoids que l’huile de coco (seule), mais la difference n’est pas tres significative ;[86] a l’interieur des cellules adipeuses individuelles, lorsqu’on les mesure par des bio-marqueurs de la lipolyse, le CLK et l’huile de coco renforcent (mutuellement) leurs effets respectifs. Comme vous pouvez le constater, le CLK et l’huile de coco augmentent de maniere synergique les effets de lipolyse de l’un et de l’autre, prevenant ainsi activement le developpement de l’obesite, mais en raison du manque de recherche “humaine” dans ce domaine et des actions pas toujours previsibles du CLK, on ne peut pas s’y fier completement. Vitamine E Une experience utilisant la vitamine E (succinate, en application topique) et l’huile de coco (myritol – 318 ; huile de coco CCT) comme base, a montre que l’huile de coco ameliorait l’absorption de la vitamine E d’environ 50% (par rapport aux autres huiles testees) ; 61,2% de la vitamine E etait dissoute dans le sang (des souris) avec le myritol 318, 43,4% avec l’huile de noix, 42,6% avec l’huile d’olive, 40,1% avec l’huile de sesame, 39,6% avec l’huile de soja, 39,4% avec l’huile de tournesol, 36% avec l’huile de carthame et 24% avec l’huile de colza. Les PTC d’huile de coco d’une autre marque (Henkel) ont un effet similaire. L’absorption de la vitamine E est donc directement affectee par les PTC eux-memes, quel que soit le fabricant de l’huile de coco. Les triglycerides a chaine moyenne de l’huile de noix de coco, contrairement a d’autres acides gras, ameliorent l’absorption de la vitamine E, une vitamine que l’on trouve a l’origine dans l’huile de noix de coco. L’huile de coco est largement utilisee dans les cosmetiques et, selon une validation de la FDA en 2007, elle est un ingredient de 626 produits cosmetiques dans 142 applications cosmetiques. Selon une evaluation de la securite de la sante humaine realisee en 1986, l’huile de coco, lorsqu’elle est appliquee sur la peau, n’est pas irritante, convient aux peaux sensibles, n’est pas toxique et provoque tres rarement des reactions allergiques legeres. Elle est toleree dans les cosmetiques jusqu’a 50% d’huile de coco (ce qui est parfaitement sur pour la sante). Ce rapport a ete approuve par un comite d’experts. Il contient une liste complete des acides gras et de ses autres ingredients, qui sont tous sans danger pour la peau (cocoate glycerol, acide lanolique, alcools, butylene glycol, etc, confirming the safety of coconut oil in cosmetic products. Polycyclic aromatic hydrocarbons (PAHs) known as carcinogens in meats that have been heavily smoked which are made in large amounts after the coconut oil is repeatedly (but not solely) heated. For rats that had been exposed for more than two times (or even) heated oil to cook, the livers began in shrinkage (shrink to smaller sizes). This isn’t only the case with coconut oil but it also happens with other oils, such as sunflower oil (when heated continuously) and soybean oil (in this instance , repeated heating could lead to atherosclerosis) and palm oil.25) The constant warming of oils (stir-frying in the same oil that’s used in restaurants) is extremely harmful for health as a result of active peroxidation of the lipids, carcinogens are released. Coconut oil isn’t as prone to this (due due to the fact that it is a rich source of antioxidants and particularly saturated fats) however it shouldn’t be heated as well.
